Chapter 02 Diagnosis · Staging · Recession · Treatment

Periodontics

The largest single diagnosis category on the OSCE — and an exam loved for its multi-answer questions where one missed pick costs the whole mark. The trick is reading BOP, CAL, and PD as a triangle, not three separate numbers.

14%
Largest Dx Category
01

Diagnosis Decision Map

Three numbers decide everything: BOP %, PD, CAL. Read them as a triangle.

Figure 1.1

The diagnostic triangle

BOP percentage? first gate < 10% HEALTH LANE Then check CAL + PD ≥ 10% INFLAMMATION LANE Gingivitis or Periodontitis CAL = 0 Intact periodontium PD ≤ 3 mm CAL present Reduced periodontium non-perio cause CAL = 0 / PD ≤ 3 Gingivitis biofilm or systemic CAL ≥ 2 sites Periodontitis → stage + grade CAL = clinical attachment loss · PD = probing depth · BOP = bleeding on probing

Diagnostic categories — full reference

Health

TypeBOPPDCALDescription
Clinical Gingival Health< 10%≤ 3 mm0No clinical inflammation
Intact Periodontium< 10%≤ 3 mm0No CAL, no recession, no periodontitis history
Reduced Periodontium (non-perio)< 10%≤ 3 mmmay existAggressive brushing, crown lengthening
Reduced Periodontium (Stable Perio Pt)< 10%≤ 4 mmmay existTreated periodontitis. No 4 mm pocket WITH BOP.

Gingivitis

TypeBOPPDCALDescription
Biofilm-induced gingivitis≥ 10%≤ 3 mm0Plaque-mediated
Mediated by systemic / local risk factors≥ 10%≤ 3 mm0Hormones (puberty/pregnancy), hyperglycemia, malnutrition; subgingival restorations, hyposalivation
Drug-influenced gingival enlargement≥ 10%≥ 3 mm0Pseudo-pocketing from nifedipine, phenytoin, cyclosporine

Periodontitis

TypeThreshold
PeriodontitisCAL at ≥ 2 non-adjacent teeth OR buccal CAL > 3 mm with PD > 3 mm at ≥ 2 teeth
M
Memory Hook · The two gates BOP < 10% = health lane. BOP ≥ 10% = inflammation lane. Then ask: any CAL? PD > 3? That's three questions in 5 seconds.
i
OSCE pattern · "Select ONE OR MORE" Multiple boxes can be correct simultaneously — e.g., "Clinical Gingival Health" + "Reduced Periodontium" + "Stable Periodontitis Patient" all check together for a treated patient.
02

Staging & Grading

Stage = "How far gone?" (worst CAL + RBL + tooth loss). Grade = "How fast?" (rate of bone loss, modified by smoking and diabetes).

Stages I → IV

I

Initial

Interdental CAL
1–2 mm
RBL
Coronal third < 15%
Tooth loss
None
Max PD
≤ 4 mm
II

Moderate

Interdental CAL
3–4 mm
RBL
Coronal 15–33%
Tooth loss
None
Max PD
≤ 5 mm
III

Severe

Interdental CAL
≥ 5 mm
RBL
Middle/apical 1/3
Tooth loss
≤ 4 teeth
Complexity
PD ≥ 6 · vertical > 3 · furcation II/III
IV

Advanced

Interdental CAL
≥ 5 mm
RBL
Middle/apical 1/3
Tooth loss
≥ 5 teeth
Complexity
Mob ≥ 2 · bite collapse · < 20 teeth

Grades A · B · C

A: SlowB: ModerateC: Rapid
RBL or CAL over 5 yrNone< 2 mm≥ 2 mm
% RBL / age (indirect)< 0.250.25 – 1.0> 1.0; or destruction exceeds biofilm
SmokingNon-smoker< 10 cig/day≥ 10 cig/day
DiabetesNormoglycemicHbA1c < 7.0%HbA1c ≥ 7.0%
M
Memory Hook · Two questions Stage = "How far gone?" Grade = "How fast?" RBL/age: <0.25 slow → 0.25-1 moderate → >1 rapid.
03

Recession (Cairo) & Necrotizing Diseases

Cairo classifies recession by interproximal CAL. The number tells you whether root coverage is even possible.

Figure 3.1

Cairo Recession Types — root coverage prognosis

RT 1 No interproximal CAL 100% coverage possible RT 2 Interprox CAL ≤ buccal Partial coverage RT 3 Interprox CAL > buccal Full coverage NOT possible gingiva recession (buccal) interproximal loss
M
Memory Hook · The comparison Compare interproximal CAL to buccal CAL: none → RT1 (full cover possible). buccal → RT2 (partial). > buccal → RT3 (no full coverage).

Necrotizing diseases & perio-systemic manifestations

TypeDescriptionInitial Management
Periodontitis as Manifestation of Systemic DiseaseDiabetes, HIV, Down syndrome → influence inflammationSRP + CHX + systemic abx if indicated
Necrotizing GingivitisAcute gingival necrosis, bleeding, pain. Punched-out papillae.Gentle debridement + CHX + systemic abx if systemic signs
Necrotizing Periodontitis+ bone loss + halitosis. Often immunocompromised.SRP + CHX + systemic abx
Necrotizing StomatitisNecrosis beyond gingiva — soft tissue / bone.Debridement + CHX + abx + urgent medical referral
!
Trap · Necrotizing buzzwords Spontaneous bleeding + pain + halitosis + papillary necrosis (punched-out papillae). Immunocompromised history → strongly favor necrotizing diagnosis.
04

Treatment Phases

Three phases. Restorative work waits until perio is stable.

Figure 4.1

Treatment phase flow

PHASE 1 Non-surgical Risk factors · OHI · SRP Caries · endo · fix restorations Extract hopeless · splint · adjust Re-eval 4–6 wk PHASE 2 Surgical Periodontal surgery Resective / regenerative Implants · bone augmentation Stable PHASE 3 Restorative + Maint. Restorations · prostho Occlusal therapy Maintenance every 3–6 mo

Surgical procedures — when to pick which

ProcedureDoes whatIndication
Coronally Positioned FlapRepositions flap to cover rootRecession with sensitivity; aesthetic root coverage
Laterally Positioned FlapSlides donor flap from neighbourIsolated recession with adequate adjacent attached gingiva
Free Gingival GraftPalatal graft to widen keratinized tissue↑ keratinized gingiva; multiple sites; less esthetic demand
Subepithelial CT GraftCT graft, bilaminar/envelope recipientGold standard for esthetic root coverage
GTR / RegenerativeMembrane → selective regenerationGrade II furcation; 2- or 3-wall vertical defects 4-5 mm
GingivectomyExcise gingiva to remove pocketDrug-induced enlargement; pseudo-pockets
Distal WedgeEliminate distal pocket on terminal molarDistal pocket on most distal tooth
Root Amputation / HemisectionRemove one root or split molarSevere perio loss / VRF / furcation on one root
FrenectomyRemove abnormal frenumHigh frenum → recession; midline diastema
Surgical Crown ExposureExpose impacted/unerupted toothFor ortho traction
Sinus LiftElevate sinus floor + graftInsufficient bone height for posterior maxillary implant
Occlusal TherapyReduce traumatic forcesAdjunct when traumatic occlusion → mobility, fremitus
05

Flap Designs

Three extraction flaps; named for their vertical incisions. More access = more verticals.

Figure 5.1

Extraction flap geometry

Envelope No vertical · 2M + 1D most surgical extractions Triangular 1 mesial vertical max 3M · deep mand impactions Rectangular 2 verticals (M + D) OAC · deep impactions · cyst access
M
Memory Hook · "E-T-R" extraction flaps Envelope (no verticals) → Triangular (one mesial vertical) → Rectangular (two verticals). More access = more verticals.

Periodontal & mucogingival flaps

FlapDescription & indication
SemilunarCurved incision in alveolar mucosa apical to gingiva. Requires adequate vestibular depth. Used in periapical/apical surgery to preserve interdental papilla.
Coronally PositionedSplit-thickness, repositioned coronally to cover recession. Requires 3 mm keratinized gingiva.
Apically PositionedRepositioned apically to expose more tooth or reduce pocket. Crown lengthening; pocket reduction; ↑ keratinized gingiva.
Laterally PositionedSlide donor tissue from adjacent tooth. Isolated recession with adequate donor attached gingiva.
06

Contributing Factors

TypeDescription
Dental biofilm / calculusBacterial biofilm → host inflammatory response (primary etiology)
Periodontal phenotypeThin → recession risk; thick → resistant
Toothbrush traumaAggressive horizontal brushing + hard bristles → recession + cervical abrasion
Self-induced traumaFingernail biting, foreign objects → localized injury
Intraoral jewelleryLip/tongue piercings → localized recession (often mandibular anterior)
CariesSubgingival caries → disrupts supracrestal attachment
Non-carious cervical lesionsAbrasion / erosion → recession + plaque retention
Tobacco use↓ bleeding (vasoconstriction!), impairs immune response, ↑ progression
Pulpal statusNecrosis → endo-perio lesion via accessory canals
Denture-induced traumaIll-fitting dentures → mechanical trauma + plaque
Traumatic occlusal forcesDoes NOT initiate periodontitis but exacerbates attachment loss when inflammation exists
Systemic factorsDiabetes, phenytoin, cyclosporine, CCBs
Pro-inflammatory cytokinesIL-6, TNF-α, PGE₂; ↑ in poorly controlled diabetes, smoking, obesity
!
Trap · The smoker's paradox A smoker may have severe periodontitis with very LITTLE bleeding due to vasoconstriction. Don't be reassured by low BOP if pockets are deep + smoking history.
Key · Occlusal trauma alone doesn't cause periodontitis It only worsens disease when biofilm-driven inflammation already exists. Always look for the inflammatory trigger first.